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Terminating hepatocyte proliferation during liver regeneration: The roles of two members of the same family (CCAAT‐enhancer‐binding protein alpha and beta) with opposing actions

George Michalopolous M.D., Ph.D.

Corresponding Author

Department of Pathology, School of Medicine, University of Pittsburgh, Pittsburgh, PA

Address reprint requests to: George Michalopoulos, M.D., Ph.D., Department of Pathology, School of Medicine, University of Pittsburgh, 200 Lothrop Street, Pittsburgh, PA 15261. E‐mail:

michalopoulosgk@msx.upmc.edu

; fax: 412‐648‐9846.
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First published: 26 July 2014
Cited by: 4

Potential conflict of interest: Nothing to report.

See Article on Page 315

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Number of times cited: 4

  • , C/EBPα‐dependent preneoplastic tumor foci are the origin of hepatocellular carcinoma and aggressive pediatric liver cancer, Hepatology, 67, 5, (1857-1871), (2018).
  • , Pattern analysis uncovers a chronic ethanol-induced disruption of the switch-like dynamics of C/EBP-β and C/EBP-α genome-wide binding during liver regeneration, Physiological Genomics, 49, 1, (11), (2017).
  • , The role of bile salts in liver regeneration, Hepatology International, 10, 5, (733), (2016).
  • , Homocysteine Induces Collagen I Expression by Downregulating Histone Methyltransferase G9a, PLOS ONE, 10, 7, (e0130421), (2015).